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Updated: June 28, 2025
In chronic tendinitis there occurs repeated attacks of inflammation wherein lameness is pronounced and there exists in reality, at such times, acute inflammation of a hypertrophic structure, where at no time does inflammation completely subside.
A halting gait may also be produced by the abnormal development of a member, or by the shortening of the leg occasioned by the loss of a shoe. For descriptive purposes lameness may be classified as true and false. True lameness is such as is occasioned by structural or functional defects of some part of the apparatus of locomotion, such as would be caused by spavin, ring-bone, or tendinitis.
Draft animals are less likely to be permanently incapacitated as a result of tendinitis, than are thoroughbreds. Likewise, one would not expect to find this affection present in heavy harness horses as frequently as in light harness animals. Mal-formation of a part, or an asymmetrical development of the body as a whole, may render an animal susceptible to certain affections which cause lameness.
Excessive strain when put upon tendons in any possible manner, such as is occasioned in running and jumping; making missteps and catching up the weight of the body with one foot, when the force thus thrown upon the supporting structure is great because of momentum gained at a rapid pace, are exciting causes of tendinitis. Symptomatology.
Some practitioners are partial to the use of the actual cautery in these cases, but it is doubtful if it is necessary to produce such a great degree of counter-irritation in cases where the subject is suffering the first attack of tendinitis.
In acute tendinitis, the subject while at rest, maintains the affected member in volar flexion because this position permits relaxation of the inhibitory apparatus, including the inflamed tendon.
We have observed this form of tuberculous disease in the gastrocnemius and in the psoas in the latter muscle apart from tuberculous disease in the vertebræ. #Tendinitis.# German authors describe an inflammation of tendon as distinguished from inflammation of its sheath, and give it the name tendinitis.
The extensor tendons, because of the nature of their function, are very seldom strained; they are often bruised and occasionally divided, but unlike this condition in the flexors, tendinitis of the extensors is of rare occurrence.
No difficulty is encountered in making a practical diagnosis in tendinitis; that is, one may fail to readily recognize the extent of the involvement as it affects the superficial flexor tendon, for instance, but this has no practical bearing on the prognosis and treatment, when existing inflammation of the deep flexor is recognized.
Being almost inelastic and not well supplied with blood, tendinous tissue is slowly regenerated, and so much time is required for complete recovery to take place in tendinitis, that affected animals seldom fully recover before they are in service or vigorously exercising at will. As a result, complete recovery is delayed or prevented.
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