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Updated: June 20, 2025
Indeed, the growth of these organisms during the ripening is extremely slow compared to the possibilities of bacterial growth that we have already noticed. Nevertheless, the bacteria do multiply in the cheese, and as the ripening goes on they become more and more abundant, although the number fluctuates, rising and falling under different conditions.
Whatever the local dietary, during thousands of years of eating that dietary natural selection prevailed; most babies that were allergic to or not able to thrive on the available dietary, died quickly. Probably of childhood bacterial infections. The result of this weeding out process was a population closely adapted to the available dietary of a particular locale.
If the stem of a plant freshly wilted from this disease be severed, the bacteria will ooze out in dirty white drops on the cut surface. =Remedial measures= entirely satisfactory for the control of bacterial wilt have not yet been worked out. The best methods to adopt at present are the following: Rotation of crops.
Bacterial infection was a threat on every inhabited world, and the viruses the tiniest of all submicroscopic organisms were the most difficult and dangerous of them all. And yet virus plagues had been stopped before, and they could be stopped again.
Even in the sprouting of seeds after they are sown in the soil bacterial life has its influence. When seeds are placed m moist soil they germinate under the influence of heat. The rich albuminous material in the seeds furnishes excellent food, and inasmuch as bacteria abound in the soil, it is inevitable that they should grow in and feed upon the seed.
This is an important line for experimenters to follow up. There is no proof that the disease is spread through seed from diseased plants. =Fusarium wilt.= This disease and the one following resemble the bacterial wilt so closely, as far as external characters go, that they are difficult to tell apart.
They had a built-in, explosive reaction to bacterial and viral toxins, and there hadn't yet been any pathogenic organism discovered to which a tormal could not more or less immediately develop antibody-resistance. So that in interstellar medicine tormals were priceless.
The bacterial germs causing enteric and relapsing fevers are now known, and have been isolated and cultivated, and the mode in which they are conveyed into the body of a previously healthy patient is ascertained. But until the past year we knew neither the parasitic germ which causes typhus fever nor the mode by which it passes from one individual to another.
Speaking generally, it may be said that dry gangrene is essentially due to a simple interference with the blood supply of a part; while the main factor in the production of moist gangrene is bacterial infection. The cardinal signs of gangrene are: change in the colour of the part, coldness, loss of sensation and motor power, and, lastly, loss of pulsation in the arteries.
They had a built-in, explosive reaction to bacterial and viral toxins, and there hadn't yet been any pathogenic organism discovered to which a tormal could not more or less immediately develop antibody resistance. So that in interstellar medicine tormals were priceless.
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